Evidence that cross-reactive immunity from common human coronaviruses can influence response to SARS-CoV-2 Immunity SARS SARSCoV2 MERS Coronavirus COVID MDPIOpenAccess _KSU harvardmed BrighamWomens HopkinsMedicine IndianaUniv
Human CoVs are enveloped ribonucleic acid viruses from the Coronaviridae family that cause influenza-like illness, pneumonia, bronchiolitis, and exacerbation of underlying lung disease. SARS-CoV-2, a novel beta CoV causing the coronavirus disease 2019 , was first detected in Wuhan, China, and has so far caused more than 757 million cases and 6.85 million deaths worldwide.
SARS-CoV, SARS-CoV-2, and MERS-CoV share a significant sequence homology and potential epitopes than can elicit adaptive immune responses. Moreover, a high level of cross-reactivity has been observed between B- and T-cell epitopes and antibodies against structural proteins of SARS-like viruses and SARS-CoV-2. Nonetheless, there is limited evidence on the cross-reactive immunity between MERS-CoV and SARS-CoV-2.
Immunology eBook Compilation of the top interviews, articles, and news in the last year. Download a free copy Participants without a polymerase chain reaction test and those with a known immunologic disease, malignancy, or on immunosuppressive therapy were excluded. Serum samples were collected and tested for antibodies against MERS-CoV and SARS-CoV-2 by enzyme-linked immunosorbent assay .
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