New Hope in Alzheimer’s Fight: Addressing Myelin Degradation Could Prevent Disease — New research has demonstrate |
Certain immune cells, microglia , remove amyloid plaques in the brain of the Alzheimer mouse . Degenerating myelin distracts them from doing so . Credit: Max Planck Institute for Multidisciplinary Sciences
Alzheimer’s disease progresses gradually and mainly affects the elderly. The risk of developing Alzheimer’s doubles every five years after the age of 65.“The underlying mechanisms that explain the correlation between age and Alzheimer’s disease have not yet been elucidated,” says Klaus-Armin Nave, director at the MPI for Multidisciplinary Sciences.
Their work focused on a typical feature of the disease: “Alzheimer’s is characterized by the deposition of certain proteins in the brain, the so-called amyloid beta peptides, or Aꞵ peptides for short,” states Constanze Depp, one of the study’s two first authors. “The Aꞵ peptides clump together to form amyloid plaques. In Alzheimer’s patients, these plaques form many years and even decades before the first symptoms appear.
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