New strategy could restore balance in tissues and target cells that play a role in age-related illnesses. As we age, our bodies can accumulate aging, or senescent cells, which have ceased division but don't die. These cells can lead to chronic inflammation, which in turn can contribute to the devel
Senescent cells are cells that have stopped dividing and are no longer able to perform their normal functions. They accumulate with age and have been linked to a number of age-related diseases, including arthritis, cardiovascular disease, and neurodegeneration.
Studies in mice have shown that the removal of senescent cells from aging tissues can result in a restored tissue balance and a prolonged healthy lifespan. Now, a team of researchers at Massachusetts General Hospital, a founding member of Mass General Brigham, has discovered that the immune response to a commonFor the study, which is published in, the scientists analyzed young and old human skin samples to learn more about the clearance of senescent cells in human tissue.
When they assessed how killer CD4+ T cells keep senescent cells in check, the researchers found that aging skin cells express a protein, or antigen, produced by human cytomegalovirus, a pervasive herpesvirus that establishes lifelong latent infection in most humans without any symptoms. By expressing this protein, senescent cells become targets for attack by killer CD4+ T cells.
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